One hormone. Many conditions.
We start in antenatal care, where the need is most urgent.
Every condition where hepcidin dysregulation drives treatment failure is an application.
Maternal health & PPH
Iron therapy that never absorbed is the silent upstream cause of anaemia at labour.
Malaria & HIV
Both hold hepcidin high — blocking iron in the patients most likely to be prescribed it.
Heavy menstrual bleeding
Chronic iron loss plus subclinical infection. The iron is prescribed; inflammation blocks it.
Children's anaemia
Iron given when hepcidin is high feeds pathogens instead of the child. Measuring first protects them.
Cancer anaemia
Tumour inflammation keeps hepcidin elevated. Oral iron fails, and the reason is rarely identified.
Chronic kidney disease
Impaired hepcidin clearance drives iron sequestration and ESA non-response.
Transfusion safety
Pre-transfusion profiling catches iron overload in sickle cell and thalassaemia before organ damage.
Iron genetic disorders
Haemochromatosis, IRIDA, sideroblastic anaemias — where the hepcidin pathway determines treatment.
Fortification efficacy
Population surveillance shows where national fortification programmes fail at scale.
Testing today, while the RDT is in development.
We run hepcidin testing through our partner laboratory network at a discounted rate.